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Hiperglikēmija | Hiperglikēmiskā līkne 1. Norma 2. Latentais diabētsLai novērtētu ogļhidrātu v/m pacientam, izmanto cukura slodzi – iegūto glikozes līmeņa līkni sauc par hiperglikēmosko līkni.Gaita: 1. Vesels cilvēks tukšā dūšā uzņem 1-2 g/kg cukuru, bet nevairāk kā 75 g GLU 2. GLU uzsūcas KZT -> v. porta asinīs 3. aknas daļu Glu pārvērš glikogēnā, bet ne visu hiperglikēmija 4. pacienta GLU līmenis asinīs 30-60 min laikā ceļas līdz 7,3 – 9,0 mmol/L pēc 2h norma pēc 3h GLU līmenis zemāks nekā normaHiperglikēmiskais koeficients: max pēc 60 min norma: 1,5-1,7Poshiperglikēmiskais koeficients: pēc 2h norma: 0,9-1,0 | Alimentāra hiperglikēmija | Centrāla(emocionāla) hiperglikēmija | Hiperglikēmiskās endokr. regul. traucējumi | Etioloģija: * rodas pēc GLU uzņemšanasIzmanto: * GLU slodzes testu * GLU dubutslodzes tests * GLU tolerances tests | Etioloģija: * CNS emoc. traucējums * CNS organisks bojājumsMehānisms:Kairinājums izplatās pa zemgarozu un simpātiskiem ceļiem sasniedz virsnieres un vairogdziedzeri, tā rezultātā:(↑) AKT, GK, adrenalīna, tireotoksīna daudzums, kas noved pie glikogenolīzes stimulēšanas | Etioloģija: * slimību gadījuos ar hipoinsulīnismu * GLU paaugstina hormoni (STS producē hipofīzes audzēji) | Orientējošie rādītāji: 1. ja slimniekam īslaicīgi tukšā dūšā cukurs ir līdz 4,4 mmol/L, tad viņš ir vesels un nekādu papildizmeklēšanu neveic, 2. ja tukšā dušā cukurs ir virs 4,4 mmol/L un ir sūdzības, tad veic cukura slodzi 3. ja cukurs tukšā dūšā ir vairāk par 11,1 mmol/L, slimniekam ir cukra diabēts, viņš tiek nosūtīts pie endokrinologa.Īslaicīgai hiperglikēmijai nelabvēlīgu seku nav.Ilgstoša hiperglikēmija var radīt sekojošus traucējumus: 1. hiperglikēmija nepārtrauktikairina pankreasu un provocē diabēta attīstību; 2. hiperglikēmija rada glikozūriju un izraisa organisma atūdeņošanos; 3. ogļhidrāti veido kopleksus ar olbaltumu un tie izgulsnējas a/v sieniņā, veicinot arterosklerotiskas izmaiņas. |

Hipoglikēmija | Cēloņi: | Eksogēni | Endogēni | * bads * alkohola lietošana * medikamenti (INH) * insulīna preperātu pārdozēšana * ilgstoša, intensīva fiziska slodze | * bads * malabsorbcijas sindroms – ogļhidrātu malabsorbcija KZT * aknu mazspēja – glikogēna krājumi ir mazi * vielmaiņas slimības ar GLU zudumiem vai izm. traucējumiem * endokrīnās sistēmas slimības | Hipoglikēmijas stadijas | 1.stadija – Vieglas pakāpes hipoglikēmija * „vilka” apetīte * galvassāpes * reiboņi * vājums * svīšana * muskuļu trīce * zīlīšu paplašināšanās * sirdsdarbības frekvences palielināšanās2.stadija – Vidēji smaga hipoglikēmija * redzes, runas traucējumi * afekta stāvokļa parādības * uzbudinājums3.stadija – Hipoglikēmijskā koma * iestājas 1,7 – 2,0 mmo/l * galvas smadz. garozā iestājas kavēšana * zemākajos CNS centros - uzbudinājums | Hipoglikēmiskā komaGlikozes trūkums nervu šūnās.Pazīmes: * bezsamaņa * krampji * virspusēja elpošana * auksta, mitra,bāla āda * cieti acāboli-jo samazinoties Glu līmenim EC telpā ūdens pārvietojas uz šūnu, acāboli piebriest * krampji, vēdera nesaturēšana – galvas smadzeņu garoza vairs nekontrolē zemgarozas centrus.Pirmā palīdzība hipoglikēmijas gadījumā: 1. ja slimnieks ir pie samaņas – jādod apēst kaut ko saldu 2. ja iestājusies ko,a – i/v ievada 40% GL |

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...sdfs df f sdfsd fa dfd fd fsdf sd f sdf dsf dfsdfnsd fnds fn sdf dnf sd snd dg gnf gn gfn dfng ndf ndg sgn dfgn fgn fdgnf g dfg fg fgnsf ng nfg nfg fgn fgnfg ngf fng nfg fgn fdnfg fgnf nsf nsdg fng fn fgnfg n dnf dnfgng fg ng fgn dgndf ng sg gf nsgngnf gn fngfng fng fng fng fgn fgn fgnfg ndf nf g fgn gnf gn fgn fn fgsn fn sg gng nfg fgn fgnfg nfg nfg fn nfg nfg bfdan rnt th thn rng ntg ne etr en ne gnfn fjrb r nr rer ej ww edm rne rnqwer we erne f gf df wdf df fgf g g fg g g g g g df e rf r r r e re r er r4t t t 6y y uuy uii u u i u i i i i u t df d fg fg f f. life is as real as you make it fsddgggggggggggggdsaaaaaaaaaaaaaaaaaagg ds hdn fbn fndf hddhgdgfhdsf afha fhdfhdff dbf bdf ndf asnf af ddadssdfsdf df f s df dsfdf sdfs df f sdfsd fa dfd fd fsdf sd f sdf dsf dfsdfnsd fnds fn sdf dnf sd snd dg gnf gn gfn dfng ndf ndg sgn dfgn fgn fdgnf g dfg fg fgnsf ng nfg nfg fgn fgnfg ngf fng nfg fgn fdnfg fgnf nsf nsdg fng fn fgnfg n dnf dnfgng fg ng fgn dgndf ng sg gf nsgngnf gn fngfng fng fng fng fgn fgn fgnfg ndf nf g fgn gnf gn fgn fn fgsn fn sg gng nfg fgn fgnfg nfg nfg fn nfg nfg bfdan rnt th thn rng ntg ne etr en ne gnfn fjrb r nr rer ej ww edm rne rnqwer we erne f gf df wdf df fgf g g fg g g g g g df e rf r r r e re r er r4t t t 6y y uuy uii u u i u i i i i u t df d fg fg f f.life is as real as you make it fsddgggggggggggggdsaaaaaaaaaaaaaaaaaagg ds hdn fbn fndf hddhgdgfhdsf afha fhdfhdff dbf bdf ndf asnf af ddadssdfsdf df f s df dsfdf sdfs df f sdfsd fa dfd fd...

Words: 737 - Pages: 3

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Asdfg

...Qwe D fsd dfdsd d sdsdf Sdshdjh sk Sdfsfdfdsf sdffdfsdf Sdfsd Fsd Fd Fsd fzdfjvjdfhbvzfdjh vgzdfjhvhdfjgjhfdshfhdksgdfjs hghjdfghajsgjdhfasjhfghjkjhgtuwertewry ugfdjhgspdgdfvbregb r esgbzfd hjvgdfkjgk hjkgfhjdghs djfgjhsdf Sdfds F Ds dfjhdf dfjhdf dfjh eiruyi erejdf dhdjf d djhjd hdfj dfhjhd hdfj dfjhjdhf j dhjhjd fjhdj dj j j j j j j j jj j j j j j j j hh hjhj hjh jhj F Ds Fsdfsdfsdfdsfdsf Sfds F Sd Fds F Dsds F Dsf Ds Fsfds F Ds Fsdfdsfsdfdsfsdfdsfdsfsbsdhjajhfhdsagasjkhsdfgjhafgkshdsjhvz kdjhvbzxcvjzdfhvbfjhgzbvdfgvzdfjhvbzfdjhvbzdfvfdjhgvfdbvzdfb vjfhdgbzdfjbv hgh ghgh ghg ghgh ghg gh gh g h hg h hg h h gh hg h hg g h h h h h h h h h h h h h h h h h h h h h h h h h h h hhh h h h h hhhhh hh h hhh h h hhh h hh Qwe D fsd dfdsd d sdsdf Sdshdjh sk Sdfsfdfdsf sdffdfsdf Sdfsd Fsd Fd Fsd fzdfjvjdfhbvzfdjh vgzdfjhvhdfjgjhfdshfhdksgdfjs hghjdfghajsgjdhfasjhfghjkjhgtuwertewry ugfdjhgspdgdfvbregb r esgbzfd hjvgdfkjgk hjkgfhjdghs djfgjhsdf Sdfds F Ds dfjhdf dfjhdf dfjh eiruyi erejdf dhdjf d djhjd hdfj dfhjhd hdfj dfjhjdhf j dhjhjd fjhdj dj j j j j j j j jj j j j j j j j hh hjhj hjh jhj F Ds Fsdfsdfsdfdsfdsf Sfds F Sd Fds F Dsds F Dsf Ds Fsfds F Ds Fsdfdsfsdfdsfsdfdsfdsfsbsdhjajhfhdsagasjkhsdfgjhafgkshdsjhvz kdjhvbzxcvjzdfhvbfjhgzbvdfgvzdfjhvbzfdjhvbzdfvfdjhgvfdbvzdfb vjfhdgbzdfjbv hgh ghgh ghg ghgh ghg gh gh g h hg h hg h h gh hg h hg g h h h h h h h h h h h h h h h h h h h h h h h h h h h...

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