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Chronic Traumatic Encephalopathy

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Chronic Traumatic Encephalopathy

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There is a dark cloud hanging over the world of contact sports and it is growing at an alarming rate. With the size and speed of today’s athletes, the sports of football and hockey have become more exciting, fast paced, wide open, and fun to watch. However, there is another consequence of these ever growing athletes on their sports. They have made the collisions in them increasingly more violent. The velocity that these athletes hurl themselves through the air has created an atmosphere that could not have been imagined when these sports were created. First described in the year 1928 (McKee 2010), Chronic Traumatic Encephalopathy, or CTE, is a progressive neurodegenerative disease.

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CTE is famously regarded to be the cause of retired NFL linebacker, Junior Seau’s suicide. The disease deteriorated his brain and hindered his ability to think logically. Seau is not the only retired NFL player found to have had CTE through autopsy following their death. Mike Webster was the first football player found to have CTE, when scientists found the characteristic buildup of the tau protein in his brain. Another significant find

in CTE affected brains such as his, includes the shrinkage of the hippocampus, the part of the brain responsible for memory and thinking processes. The disease was originally noticed in boxers, first being called “punch drunk.” These boxers were described as exhibiting “cuckoo” and “goofy” characteristics. The observation in boxers led to a term meaning dementia of a fighter – “dementia pugilistica” which was later renamed its current name, Chronic Traumatic Encephalopathy, in the 1960s (Saulle 2012).

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It’s been confirmed that the cause of CTE is repetitive head trauma but it is unclear whether or not it can be brought on by a single

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