APRIL 2012 An Adaptive Differential Evolution Algorithm With Novel Mutation and Crossover Strategies for Global Numerical Optimization Sk. Minhazul Islam, Swagatam Das, Member, IEEE, Saurav Ghosh, Subhrajit Roy, and Ponnuthurai Nagaratnam Suganthan, Senior Member, IEEE Abstract—Differential evolution (DE) is one of the most powerful stochastic real parameter optimizers of current interest. In this paper, we propose a new mutation strategy, a fitnessinduced parent selection scheme for the binomial
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A Review of Colorectal Cancer The colon and rectum both preform major functions daily in the digestion system. The colon absorbs water and converts the leftover food into indigestible fecal matter. The rectum stores the fecal matter until it is time to dispose of it. Colorectal cancer occurs when the cells of these two organs become mutated and divide rapidly. Unless colorectal cancer is detected early, more often than not, it becomes deadly. When food enters the digestion system, it travels
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incurable and death always occurred because inheritance patterns had not been established, leading back to more deaths. But in all actuality, the survival rate will drop with every decade of life when the patients have the genomic mutation. The genomic mutation is a gene mutation within every cell of the individual’s body. Those patients with that type of cancer have either bilateral disease or unilateral-multifocal disease. Those individuals have a predisposition for developing second cancers later
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tissue. Significant advances in understanding the cellular basis of cancer and the underlying biological mechanisms of tumour has been vastly improved in the recent years (Jiang et al. 1994). Cancer is a genetic disease which requires a series of mutation during mitosis to develop, its characteristics can be associated with their ability to grow and divide abnormal cells uncontrollable while in the mean time invade and cause nearby blood vessels to serve its need. Even though many people are affected
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Student Examination into the Causes, Treatment, and Prognosis of Osteogenesis Imperfecta Anitra Swann Baltimore City Community College Professor McNair AH 130 October 18, 2011 Abstract Osteogenesis Imperfecta is a heritable disorder of bone formation resulting in low bone mass and a propensity to fracture. It exhibits a broad range of clinical severity, ranging from multiple fracturing in utero and perinatal deaths to normal adult stature and a low fracture incidence. The disorder is currently
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been of assistance in developing of DNA based methods that are used in determining blood group genotype. Inherited blood group antigens have been described on human red cells surface. They were revealed by detecting of antigen specific anti bodies, which are mostly found in human serum. Very few of these blood group antigens are actually polymorphic in the world. This means that they have alternate alleles at great levels that can be maintained by a mutation that has to be recurrent. A few
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only consist in plants, animal breeding and fertilization of crops it also includes polyploidy (organism which two sets of homologous chromosomes), mutagenesis (process in which the organisms genetic material is changed in sable manner that causes mutation), and cell fusion techniques which does not involve recombinant nucleic acid or genetically modified organism in the process (Wikipedia). Cloning and stem cells also do take a part in Genetic Engineering (Wikipedia). Advances in Genetic Engineering
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under a circle to and from a Mandol_SK user. 2. SDO User is authorised/capable of - Finalization of Partition process - Approving registered deed on either online or offline method. 3. SDC User is authorised/capable of - Finalization of Mutation and Misc. Case process - Changing Land Class under Misc. Case process - Changing Land Area under Misc. Case process - Re-building/re-entering of Left Out records of
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Dr. Warren Tay, an ophthalmologist, was the first to characterize one of the tell tale signs of the disease, the retina of the eye has a cherry red spot. A New York, neurologist, Dr. Bernard Sachs provided the first description of the biological mutations with Tay-Sachs disease. Tay-Sachs is a progressive neurodegenerative disorders. The individuals diagnosed with the disease experience a steady loss of the cell body in the central nervous system until the disease ends the patient’s life, usually
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Therefore, mitochondrial diseases are caused by mutations in wither mtDNA or nuclear DNA (Overview of Mitochondrial Diseases). A male does not transmit the mtDNA pathogenic variant to his offspring. A female harboring a herteroplasmic mtDNA single nucleotide variant may transmit a variable amount of mutant
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