Obesity isn’t classified as an eating problem anymore. It is more than that a person’s biology plays a big roll. Cells and hormones play a big roll on how big you are and how your body regulates energy. Food equals energy and the body stores that energy in fat cells and uses the energy as needed. The metabolism breaks down that food and uses the energy generated from that food for everyday activities such as walking, talking, thinking. The faster ones metabolism is the more efficient they are at breaking down food and using the energy. The slower the metabolism the more energy gets stored and gets turned into fat cells and not used. The hormone leptin, which is produced in fat cells, sends signals to the brain telling it that its levels are dropping which means the cells are reducing. When this occurs the brain processes that information as starvation and sends signals back to slow down the metabolism and store as much energy as possible. There are people who eat less than other but still gain weight. That’s a biological problem. There are areas in the brain research has found that if damaged can lead to obesity. One such area is the ventromedial hypothalamus and the surrounding axons. If this area in the brain is damaged at then this can lead to overeating and more storing of fat cells. Research conducted on rats at the University of Nebraska looked at this area in the brain and how it affects food intake and body weight. With damage to that area the rat nearly tripled in size in the cores of about sixty days. Food intake does not always correspond to body weight. It is possible to excessively eat and not gain weight. It just matters how active one is and how efficient the metabolism is. There are other areas in the brain located in the prefrontal cortex that also can affect body weight. Those areas are responsible for food-seeking behaviors. If one is always